References
Albert, M. A., Danielson, E., Rifai, N., et al. (2001).
Effect of statin therapy on C-reactive protein levels: The
Pravastatin Inflammation/CPR Evaluation (PRINCE): A randomized
trial and cohort study. JAMA, 286: 64-70.
Abramson, J. L., & Vaccarino, V. (2002). Relationship
between physical activity and inflammation among apparently
healthy middle-aged and older US adults. Arch Intern Med.
162(11): 1286-1292.
Ballantyne, C., et al. (2003). Lipoprotein-Associated
Phospholipase A2 and Risk for Incident Coronary Heart
Disease in Middle-Aged Men and Women in the ARIC Study.
Unpublished data. Presented at the American College of
Cardiology on May 2, 2003.
Castelli, W, et al. (1996). Lipids, risk factors and
ischemic heart disease. Atherosclerosis 124(Suppl):S1-9.
Church, T. S., Bartlow, C. E., Earnest, C.P., et al.,
(2002). Associations between cardiorespiratory fitness
and C-reactive protein in men. Arterioscler Thromb Vasc
Biol. 22(11): 1869-1876.
Dada N, et al. (2002). Lp-PLA2: an emerging biomarker
of coronary heart disease. Expert Rev. Mol. Diagn. 2(1),
17-22.
Esposito, K., Pontillo, A., DiPalo, C, et al. (2003).
Effect of weight loss and lifestyle changes on vascular
inflammatory markers in obese women. JAMA, 289(14): 1799-1804.
Expert Panel on Detection, Evaluation, and Treatment
of High Blood Cholesterol in Adults. Executive summary
of the Third Report of the National Cholesterol Education
Program (NCEP). Expert Panel on Detection, Evaluation,
and Treatment of High Blood Cholesterol in Adults (Adult
Treatment Panel III). JAMA, 2001, 285: 2486-2497.
Festa, A., D’Agostine, R., Jr., Tracy, R. P., &
Huffier, S. M. (2002). C-reactive protein is more strongly
related to post-glucose load glucose than to fasting glucose
in non-diabetic subjects: The Insulin Resistance Atherosclerosis
Study. Diabetic Med, 19(11): 939-943.
Ford, E. S. (2002). Does exercise reduce inflammation?
Physical activity and C-reactive protein among U.S. adults.
Epidemiology, 13(5): 561-568.
Freinkel, S. (2003, July/August). CRP screening may be
the best weapon against heart disease since cholesterol
testing. Health: 45-50.
Gabay, C., & Kushner, I. (1999). Acute-phase and
other systemic responses to inflammation. N Engl J Med,
340: 448-454.
Huerta, M. G., & Nadler, J. L. (2002). Role inflammatory
pathways in the development and cardiovascular complications
of type 2 diabetes. Curr Diab Rep, 2(5): 396-402.
Isasi, C. R., Deckelbaum, R. J., Tracy, R. P., et al.
(2003). Physical fitness and C-reactive protein level
in children and young adults: The Columbia University
BioMarkers Study. Pediatrics, 111(2): 332-338.
Keller, K. B., & Lemberg, L. (2003). Obesity and
the metabolic syndrome. Am J Crit Care, 12(2): 167-170.
King, D. E., Carek, P., Mainous, A. G., 3rd., Pearson,
W. S. (2003). Inflammatory markers and exercise: Differences
related to exercise type. Med Sci Sports Exerc, 35(4):
575-581.
Libby, P., Ridker, P. M., & Maseri, A. (2002). Inflammation
and atherosclerosis. Circulation, 105: 1135-1143.
MacPhee, C, et al. (2001). Lipoprotein-associated phospholipase
A2: a potential new risk factor for coronary artery disease
and a therapeutic target. Current Opinion in Pharmacology,
1: 121-125.
Mann, P. J., Williams, D. P., Snow, C.M., & Wonder,
R. C. (2003). Physical activity, body fat, and serum C-reactive
protein in postmenopausal women with and without hormone
replacement. Am J Human Biol, 15(1): 91-100.
Mayo Clinic Newsletter. (2003). Novel risk factors: Identifying
new culprits in heart disease. Retrieved June 27, 2003:
http://www.mayoclinic.com/invoke.cfm?id=HB00031.
MEDLINEPlus Medical Encyclopedia. C-reactive protein.
Retrieved June 27, 2003: http://www.nlm.nih.gov/medlineplus/ency/article/00356.htm.
Mold, C., Gewurz, H., & DuClos, T.W. (1999). Regulation
of complement activation of C-reactive Protein. Immunopharmocology,
42: 23-30.
Packard CJ, et al. (2000). Lipoprotein-associated phospholipase
A2 as an independent predictor of coronary heart disease.
West of Scotland Coronary Prevention Study Group. N Engl
J Med., 343(16):1179-82.
Pearson, T. A., (2002). New tools for coronary risk assessment:
What are their advantages and limitations? Circulation,
105: 886-892.
Pearson, T.A., Mensah, G. A., et al. (2003). Markers
of inflammation and cardiovascular disease (Application
to clinical and public health practice; A statement for
healthcare professionals from the Centers for Disease
Control and Prevention and the American Heart Association).
Circulation, 107: 499-511.
Pepys, M. B., & Hirshcfield, G. M. (2003). C-reactive
protein: A critical update. J. Clin Invest. 111(12): 1805-1812.
Reynolds, G. D., & Vance R. P. (1987). C-reactive
protein immunohistochemical localization in normal and
atherosclerotic human aortas. Arch Pathol Lab Med, 111:
265-269.
Ridker, P. M. (2003). Clinical application of C-reactive
protein for cardiovascular disease detection and prevention.
Circulation, 107: 363-369.
Ridker, P, M. (2001). Should statin therapy be considered
for patients with elevated C-reactive protein? The need
for a definitive clinical trail. Eur Heart J., 22: 2135-2137.
Ridker, P. M., Buring, J. E., Cook, N.R., & Rifai,
N. (2003). C-reactive protein, the metabolic syndrome,
and risk of incident cardiovascular events: An 8-year
follow-up of 14,719 initially healthy American women.
Circulation, 107: 391-397.
Ridker, P. M., Hennekens, C. H., Buring, J. E., et al.,
(2000). C-reactive protein and other markers of inflammation
in the prediction of cardiovascular disease in women.
N Engl J Med, 342: 836-843.
Ridker, P. M., Rifai, N. Rose, L., et al. (2002). Comparison
of C-reactive protein and low-density lipoprotein cholesterol
levels in the predication of first cardiovascular events.
N Engl J Med., 347: 1557-1565.
Ridker, P. M, Rifai, N., Pfeffer, M., et al. (1999).
Long-term effects of pravastatin on plasma concentration
of C-reactive protein. Circulation, 100: 230-235.
Rothenbacher, D., Hoffmeister, A., Brenner, H. Koenigh,
W. (2003). Physical activity, coronary heart disease,
and inflammatory response. Arch Intern Med, 163(10): 1200-1205.
Saito, I., Yonemasu, K, & Inami, F. (2003). Association
of body mass index, body fat, and weight gain with inflammation
markers among rural residents in Japan. Circulation, 67:
323-329.
Schwartz, R. S., Bayes-Genis, A., Lesser, J. R., et al.
(2003). Detecting vulnerable plaque using peripheral blood:
Inflammatory and cellular markers. J Interv Cardiol, 16(3):
231-242.
Suckling, K, et al. (2002). Lipoprotein-associated phospholipase
A2: a target directed at the atherosclerotic plaque. Expert
Opn. Ther. Targets, 6 (3): 1-6.
Tillett, W. S., & Francis T., Jr. (1930). Serological
reactions in pneumonia with non-protein somatic fraction
of pneumonococcus. J Exp Med, 52: 561-571.
Tsimihodimos T, et al. (2003). Fenofibrate induces HDL-associated
PAF-acetylhydrolase but attenuates enzyme activity associated
with apo B-containing lipoproteins. Journal of Lipid Research,
44(5): 927-34.
Verma, S., Wang, C-H., Weisel, R. D. et al. (2003). Hyperglycemia
potentiates the proatherogenic effects of C-reactive protein:
reversal with rosiglitazone. Journal of Molecular and
Cellular Cardiology, 35: 417-419.
U.S. Public Health Service. (1964). Smoking and Health:
Report of the Advisory Committee to the Surgeon General
of the Public Health Service. PHS Publication No. 1103.
Yeh, E. T. H., Anderson, H. V., Pasceri V., et al. (2001).
C-reactive protein: Linking inflammation to cardiovascular
complications. Circulation, 104: 974-975.
Yeh, E. T. H., & Willerson, J. T. (2003). Coming
of age of C-reactive protein: Using inflammation markers
in cardiology. Circulation, 107: 370-372.
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